NAVLE Multisystemic

Feline Shock Study Guide

Shock is defined as a state of inadequate cellular energy production due to insufficient oxygen delivery to tissues. In cats, shock represents a critical emergency with unique species-specific manifestations that differ significantly from dogs.

Overview and Clinical Importance

Shock is defined as a state of inadequate cellular energy production due to insufficient oxygen delivery to tissues. In cats, shock represents a critical emergency with unique species-specific manifestations that differ significantly from dogs. Understanding these feline-specific presentations is essential for NAVLE success and clinical practice.

The feline response to shock is characterized by the classic triad of bradycardia, hypotension, and hypothermia, which contrasts sharply with the tachycardic compensatory response typically seen in dogs. This unique presentation occurs because cats have vagal fibers located close to sympathetic fibers, and hypotension can stimulate both pathways simultaneously.

High-YieldIf a cat's heart rate is less than 160 bpm in a veterinary clinic setting, consider shock until proven otherwise. Normal stressed cats in the clinic typically have heart rates of at least 180 bpm.
Type of Shock Mechanism Common Causes in Cats
Hypovolemic Decreased intravascular volume leading to reduced preload and cardiac output Hemorrhage (trauma, GI bleeding), vomiting/diarrhea, pancreatitis, hepatic lipidosis, burns
Cardiogenic Primary cardiac pump failure with normal to increased intravascular volume Hypertrophic cardiomyopathy (HCM), dilated cardiomyopathy, arrhythmias, pericardial effusion
Distributive Maldistribution of blood flow due to inappropriate vasodilation despite adequate volume Sepsis/SIRS, anaphylaxis, neurogenic (spinal trauma)
Obstructive Physical obstruction to blood flow causing decreased cardiac output Feline aortic thromboembolism (FATE), cardiac tamponade, tension pneumothorax

Pathophysiology of Shock

At the cellular level, shock occurs when oxygen delivery (DO2) is insufficient to meet tissue oxygen consumption (VO2). Oxygen delivery depends on three critical factors: cardiac output (CO), hemoglobin concentration (Hb), and arterial oxygen saturation (SaO2).

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