Patellar luxation is one of the most common orthopedic conditions in dogs, characterized by displacement of the patella from the femoral trochlear groove.
Overview and Clinical Importance
Patellar luxation is one of the most common orthopedic conditions in dogs, characterized by displacement of the patella from the femoral trochlear groove. This condition represents a significant category of musculoskeletal disease on the NAVLE and is frequently encountered in clinical practice.
Patellar luxation can be medial (toward midline), lateral (away from midline), or bidirectional. Medial patellar luxation is approximately 12 times more common in small breed dogs compared to large breeds, while lateral luxation is more commonly seen in large and giant breeds.
| Luxation Type |
Predisposed Breeds |
Typical Body Size |
| Medial Patellar Luxation |
Poodle, Pomeranian, Yorkshire Terrier, Chihuahua, French Bulldog, Lhasa Apso, Cavalier King Charles Spaniel, Bichon Frise, Pug, Bulldog, West Highland White Terrier, Jack Russell Terrier, Shih Tzu |
Toy and small breeds (under 10 kg) |
| Lateral Patellar Luxation |
St. Bernard, Great Dane, Irish Wolfhound, Chinese Shar-Pei, Flat-Coated Retriever, Akita |
Large and giant breeds (over 25 kg) |
Epidemiology and Breed Predisposition
Demographics
Sex predisposition: Females are 1.5 times more likely to be affected than males across all breeds.
Neuter status: Neutered dogs have approximately 3 times the odds of developing patellar luxation compared to intact dogs.
Age of diagnosis: Most luxations are diagnosed in young adults, typically within 3 years of age, though the condition is congenital or developmental in nature.
Bilateral occurrence: Reported in 20 to 52 percent of affected dogs. Bilateral luxation occurs in 46 percent of small dogs and 36 percent of large dogs.
Breed-Specific Patterns
NAVLE TipWhen you see a small breed dog (especially Pomeranian, Chihuahua, or Yorkshire Terrier) with intermittent hindlimb lameness described as 'skipping,' think medial patellar luxation first. The classic presentation is a dog that holds the leg up for a few steps, then extends it backward to 'pop' the patella back into place before resuming normal gait.
| Anatomic Location |
Deformity |
Clinical Significance |
| Hip |
Coxa vara (decreased femoral neck angle), decreased femoral neck anteversion |
Initiates malalignment cascade; creates laxity in extensor mechanism |
| Distal Femur |
Distal femoral varus, femoral torsion, hypoplastic medial femoral condyle |
Most common deformity; severity increases with luxation grade |
| Trochlear Groove |
Shallow or absent trochlear groove, poorly developed or absent medial trochlear ridge |
Results from lack of normal patellar pressure on articular cartilage during growth |
| Proximal Tibia |
Medial displacement of tibial tuberosity, internal rotation of tibia at stifle, proximal tibial varus, external tibial torsion |
Perpetuates malalignment; correction often requires tibial tuberosity transposition |
| Patella |
Hypoplasia of patella, patella alta or patella baja |
Patella alta (high-riding) common in Akitas; patella baja (low) in chondrodystrophic breeds |
Pathophysiology and Etiology
Primary Etiology
The vast majority of patellar luxations are congenital and developmental in origin, with a strong hereditary component. Although the exact mode of inheritance is not fully understood, genomic studies have identified chromosomal anomalies in specific breeds such as the Dutch Flat-Coated Retriever and Pomeranian.
Traumatic patellar luxation can occur following blunt trauma to the stifle joint, causing tearing or stretching of the joint capsule and retinacular structures. This is less common and typically has acute onset associated with a specific injury.
Quadriceps Mechanism Malalignment
The fundamental problem in patellar luxation is malalignment of the quadriceps extensor mechanism. This mechanism consists of the quadriceps muscle group, patella, trochlear groove, patellar ligament, and tibial tuberosity. Current theory suggests the pathology begins at the hip joint level, with abnormalities cascading distally.
The quadriceps angle (Q-angle) is formed between the axis of the rectus femoris muscle and the patellar tendon. Small increases in the Q-angle produce dramatic increases in medially directed vector forces, triggering progressive limb deformities during growth.
Associated Skeletal Deformities
Multiple skeletal abnormalities develop secondarily to the abnormal forces exerted during growth:
Exam Focus: The NAVLE loves to test the concept that skeletal deformities in patellar luxation develop SECONDARILY to the malalignment, not as the primary cause. The abnormal forces during growth remodel the developing bones. Remember: it's not that the shallow groove causes luxation; rather, the luxation prevents normal groove development.
| Grade |
Physical Examination Findings |
Clinical Signs and Skeletal Changes |
| I |
Patella can be manually luxated with digital pressure (usually with stifle in extension) but returns immediately to normal position when pressure is removed |
Clinical Signs:
Usually asymptomatic; occasional 'skipping' lameness. Mild internal tibial rotation and hock abduction may be observed.
Skeletal Changes:
Minimal deformity; shallow trochlear groove may be present |
| II |
Patella luxates spontaneously with stifle flexion or extension and tends to remain luxated; can be manually reduced and remains in the trochlear groove most of the time |
Clinical Signs:
Intermittent, non-painful 'skipping' lameness; dog holds leg up for few steps then extends backward to reduce patella. May progress to grade III.
Skeletal Changes:
Tibial torsional deformity typical; shallow trochlear groove; early osteoarthritis may develop |
| III |
Patella is permanently luxated but can be manually reduced temporarily with digital pressure; spontaneously reluxates when pressure is released |
Clinical Signs:
Persistent lameness; outward bowing of stifles (genu varum) in medial luxation or knock-kneed appearance (genu valgum) in lateral luxation; abnormal 'crouched' gait if bilateral
Skeletal Changes:
Severe femoral and tibial deformities; shallow or absent trochlear groove; 60 to 90 degree tibial rotation; significant osteoarthritis |
| IV |
Patella is permanently luxated and cannot be manually reduced; trochlear groove often completely absent or convex rather than concave |
Clinical Signs:
Severe, debilitating lameness; inability to fully extend stifle due to muscle contracture and soft tissue changes; marked bowlegged or knock-kneed conformation; dog may walk on forelimbs with hind limbs semiflexed
Skeletal Changes:
Severe skeletal deformities including coxa vara, distal femoral varus greater than 30 degrees, complete displacement of quadriceps mechanism; advanced osteoarthritis; irreversible soft tissue contracture |
Clinical Grading System
Patellar luxation is graded on a four-point scale based on physical examination findings. The grading system is crucial for determining appropriate treatment and prognosis.
High-YieldThe key distinction for the NAVLE is that grades I and II luxate intermittently (patella can return to normal), while grades III and IV are permanently luxated (patella is always out). Grade II is 'in most of the time,' grade III is 'out most of the time.' Remember that correlation between grade and clinical signs is NOT strong–some grade I large breed dogs are very lame, while some grade III small breed dogs show minimal signs.
| Projection |
Key Findings |
What to Look For |
| Mediolateral |
Patella superimposed on femoral condyles in backward position when luxated; osteoarthritis; meniscal mineralization |
Patellar position relative to trochlear groove; degree of osteoarthritis; joint effusion |
| Craniocaudal |
Patella medial or lateral to distal femur; varus or valgus deformity; medial displacement of tibial tuberosity |
Direction of luxation; femoral and tibial alignment; position of tibial tuberosity |
| Skyline (Tangential) |
Trochlear groove depth; integrity of trochlear ridges; presence of erosions |
Normal groove should accommodate approximately 50 percent of patella depth; assess for flattening or complete absence |
| VD Pelvis |
Coxa vara; femoral neck anteversion angle; overall limb alignment |
Hip conformation; proximal femoral deformities that initiate malalignment cascade |
Diagnostic Approach
Physical Examination Technique
Gait evaluation: Observe patient at walk and trot. Classic finding is intermittent 'skipping' lameness where the dog holds the affected limb up for a few steps. May see dog extend leg backward in attempt to reduce patella. Assess for bowlegged (genu varum) or knock-kneed (genu valgum) conformation.
Standing examination: Begin with patient standing. Palpate for joint effusion and periarticular soft tissue thickening. Locate tibial tuberosity first, then follow patellar ligament proximally to identify patella. If patella is already luxated in standing position, assign minimum grade III.
Manipulation technique: Best performed with patient in lateral recumbency to reduce muscle tension. Isolate patella between thumb and index finger of one hand. With other hand, grasp hock or foot and manipulate leg. For medial luxation: extend stifle and twist leg internally while pushing patella medially. For lateral luxation: extend stifle and twist leg externally while pushing patella laterally. Assess ease of luxation, ability to reduce, crepitus, range of motion, and presence of drawer sign (to rule out concurrent cranial cruciate ligament disease).
Additional palpation: Palpate trochlear groove depth if possible. Assess for tibial tuberosity deviation and limb torsion or angulation. Note muscle atrophy, especially of quadriceps.
Diagnostic Imaging
Radiographic evaluation is essential for confirming diagnosis, assessing skeletal deformities, and evaluating degenerative joint disease. A minimum database includes:
- Mediolateral and craniocaudal projections of the stifle
- Ventrodorsal view of the pelvis
- For high-grade luxations: orthogonal views of entire femur and tibia to assess rotational and angular deformities
Radiographic findings:
Advanced imaging: Computed tomography (CT) is increasingly used for preoperative planning, especially in high-grade luxations requiring corrective osteotomies. CT allows accurate measurement of femoral and tibial torsion, femoral varus angle, and other deformities. Three-dimensional reconstructions can be created for surgical planning.
Concurrent Conditions
Cranial cruciate ligament (CrCL) disease: Concurrent CrCL rupture has been reported in 13 to 41 percent of dogs with medial patellar luxation. Risk factors include age, higher grade of luxation, and advanced degenerative joint disease. The CrCL should be systematically assessed during orthopedic examination using cranial drawer and tibial compression tests.
The mechanism linking MPL and CrCL disease is thought to be increased strain on the ligament due to anatomic abnormalities associated with patellar luxation, leading to progressive degeneration.
NAVLE TipOn the NAVLE, if you see a middle-aged dog with patellar luxation presenting with acute-onset severe lameness, think concurrent cranial cruciate ligament rupture. The chronic joint instability from MPL predisposes to CrCL degeneration. Always check for cranial drawer sign in MPL cases.
| Treatment Component |
Recommendations |
| Pain Management |
NSAIDs (carprofen, meloxicam) for episodes of lameness. Tramadol or gabapentin for additional analgesia if needed |
| Physical Rehabilitation |
Quadriceps strengthening exercises, range of motion exercises, hydrotherapy, massage therapy. Goal is to enhance quadriceps mechanism function |
| Weight Management |
Essential to reduce stress on stifle joint. Maintain ideal body condition score of 4 to 5 out of 9 |
| Activity Modification |
Avoid jumping, stairs, and sudden direction changes. Leash walks on flat surfaces. Non-impact exercise preferred |
| Joint Support |
Glucosamine and chondroitin sulfate supplementation. Polysulfated glycosaminoglycans may help slow osteoarthritis progression |
Treatment Options
Conservative Management
Indications: Grade I patellar luxation in asymptomatic dogs or dogs with infrequent, mild lameness. Some Grade II cases with minimal clinical signs.
Prognosis with conservative management: Many Grade I dogs remain comfortable without surgery. Grade II may progress to Grade III due to progressive erosion of trochlear ridge and chronic degenerative changes. Monitor closely for progression.
Surgical Management
Indications: Grades II, III, and IV with clinical lameness or functional impairment. Some Grade II cases may be managed conservatively if infrequently symptomatic. Young dogs benefit from early surgery to prevent progressive skeletal deformity and osteoarthritis. The decision to operate on Grade II should be based on frequency and severity of clinical signs.
Soft Tissue Procedures
Soft tissue techniques address imbalanced forces on the patella but are rarely sufficient alone. Typically used as adjuncts to osseous procedures.
Osseous Procedures
Osseous techniques form the cornerstone of surgical correction. Most commonly combined procedures are used.
Tibial Tuberosity Transposition (TTT):
TTT is the single most important procedure for correcting patellar luxation. Failure to perform TTT is the most common cause of surgical failure. Performed in 71 percent of small breed cases and 86 percent of large breed cases.
Technique: Osteotomy of tibial tuberosity performed with osteotome or oscillating saw, leaving distal periosteal attachment intact. Tuberosity is transposed laterally (for MPL) or medially (for LPL) until quadriceps-patella-patellar tendon forms straight line. Fixed with Kirschner wires (0.035 to 0.062 inch diameter) and figure-of-eight tension band wire (18 to 22 gauge). Two pins provide superior stability. Pins are bent proximally and cut 3 mm beyond bend for later removal.
Critical considerations: In large, active dogs, lag screw or supplemental tension band wire is critical to prevent fixation breakdown. Tuberosity may also be moved distally to create appropriate patellar tendon tension. Adequate fragment size essential to accept implants.
Trochleoplasty (Sulcoplasty):
Techniques to deepen trochlear groove. Goal is groove deep enough to embed approximately 50 percent of patella above trochlear ridges. All techniques involve some degree of cartilage damage.
Evidence for trochleoplasty: Studies show significantly lower reluxation rates when trochleoplasty is combined with TTT compared to TTT alone. Reluxation rates: 6 to 8 percent with trochleoplasty versus 19.8 percent without. Performing trochleoplasty provides a 5.1-fold reduction in reluxation risk.
Corrective Osteotomies:
Required for significant femoral or tibial malalignment, particularly in large breed dogs and high-grade luxations. Common procedures include distal femoral osteotomy (for femoral varus) and tibial derotational osteotomy (for excessive tibial torsion). Require advanced imaging (CT) for accurate preoperative planning and measurement of deformities. Fixed with plates and screws.
High-YieldFor the NAVLE, remember that most patellar luxation surgeries require COMBINATION of techniques. The classic approach is: medial desmotomy plus lateral imbrication plus tibial tuberosity transposition plus trochleoplasty. TTT alone has high failure rate. Think of it as addressing all components of malalignment: release tight side, tighten loose side, realign tibial tuberosity, deepen groove.
Postoperative Management
| Procedure |
Technique |
Indication |
| Medial or Lateral Desmotomy (Release) |
Incision of retinaculum on same side as luxation to release tight tissues. May include joint capsule incision |
Releases tension pulling patella toward luxation. Medial release for MPL, lateral for LPL |
| Lateral or Medial Imbrication (Tightening) |
Sutures placed to tighten capsule and retinaculum on opposite side from luxation using vest-over-pants or mattress pattern |
Creates restraint opposite to luxation. Lateral imbrication for MPL, medial for LPL. Avoid excessive tightening |
| Antirotational Suture |
Fabellotibial suture from lateral fabella to tibial crest to create external tibial rotation (MPL) or from medial fabella to tibial crest (LPL) |
Repositions tibial tuberosity by rotating tibia. Alternative to tibial tuberosity transposition in select cases |
| Muscle Release or Transposition |
Dissection of rectus femoris from joint capsule and femur; or transposition of rectus femoris or cranial sartorius origin to more lateral position |
Relieves medially directed tension on patella. Used in severe MPL when other soft tissue procedures insufficient |
Complications and Prognosis
Surgical Complications
Overall complication rate: 18.5 to 29 percent. Most common complications are implant-associated problems and patellar reluxation.
Prognosis
Overall prognosis: Favorable for grades I, II, and III. Excellent to good outcome in 94 percent of surgically treated cases. Fair to poor outcome in 6 percent.
Grade-specific outcomes:
- Grade I: Excellent; most dogs remain asymptomatic with or without surgery
- Grade II: Excellent; 100 percent success rate in some studies when surgery performed
- Grade III: Good to excellent; approximately 11 percent recurrence rate
- Grade IV: Guarded, especially in young dogs; 36 percent reluxation rate due to severe skeletal deformities
Factors affecting prognosis:
- Body weight: Increasing weight associated with higher complication risk
- Age at surgery: Earlier intervention prevents progressive deformity
- Completeness of correction: Addressing all components of malalignment improves outcome
- Concurrent CrCL disease: Worsens prognosis; requires simultaneous or staged treatment
- Large breed dogs: May have less favorable prognosis due to additional orthopedic conditions like hip dysplasia
Remember: P.A.T.E.L.L.A. for Patellar Luxation Prognosis - Progression worsens without surgery, Age at surgery matters (earlier is better), Trochleoplasty reduces reluxation significantly, Early osteoarthritis develops without treatment, Large breeds face more challenges, Luxation grade correlates with outcome, Address ALL components of malalignment for success.
| Technique |
Procedure |
Advantages and Disadvantages |
| Abrasion Trochleoplasty |
Complete removal of articular cartilage and several millimeters of subchondral bone using high-speed burr, rongeur, or rasp |
Disadvantages:
Complete loss of hyaline cartilage; subsequent patellar erosion; considered antiquated technique. Rarely used today |
| Trochlear Chondroplasty |
In dogs younger than 6 months: articular cartilage incised and elevated, subchondral bone removed with curette or rasp, cartilage replaced |
Advantages:
Preserves articular cartilage if performed in immature dogs
Disadvantages:
Limited to very young patients |
| Trochlear Wedge Recession |
V-shaped osteochondral wedge removed with saw, additional subchondral bone removed, wedge replaced in recessed position. Held by press-fit and patellar pressure |
Advantages:
Preserves articular cartilage; effective deepening; no internal fixation needed
Disadvantages:
Technically demanding; wedge apex angle must be maintained precisely |
| Trochlear Block Recession |
Rectangular osteochondral block removed, additional subchondral bone removed, block replaced. Slight inward angulation of cuts creates press-fit |
Advantages:
Preserves articular cartilage; studies suggest superior biomechanical properties compared to wedge recession; easier to perform
Currently preferred technique in most cases |
| Management Component |
Protocol |
| Immediate Postoperative |
Soft padded bandage for 2 to 3 days to reduce swelling and prevent self-trauma. Postoperative radiographs to confirm implant placement and patellar alignment |
| Activity Restriction |
Strict restriction for 6 to 8 weeks. Short, slow leash walks only. No running, jumping, stairs, or off-leash activity. Gradual return to normal activity after radiographic confirmation of healing |
| Pain Management |
NSAIDs continued for 10 to 14 days. Opioids (tramadol, buprenorphine) as needed for breakthrough pain in first 3 to 5 days. Gabapentin may be added for neuropathic component |
| Physical Rehabilitation |
Passive range of motion exercises 3 times daily starting 3 to 4 days postoperatively. Progress to active exercises, hydrotherapy, and strengthening as healing allows. Prevents muscle atrophy and maintains joint mobility |
| Recheck Schedule |
Two-week recheck for suture removal and wound assessment. Six to eight-week recheck with radiographs to evaluate osteotomy healing. Implant removal after 8 to 12 weeks if indicated |
| Complication |
Incidence and Risk Factors |
Management |
| Patellar Reluxation |
Most common major complication. Higher risk with increasing body weight, higher luxation grade, and severe skeletal deformities. Grade IV: 36 percent reluxation rate. Risk significantly reduced when trochleoplasty performed with TTT |
Revision surgery required. Address any uncorrected deformities. May need corrective osteotomies if not performed initially |
| Implant-Associated Complications |
Pin migration, pin breakage, seroma formation. Higher risk in large, active dogs if fixation inadequate. Occurs in 10 to 15 percent of cases |
Pin removal if migration occurs. If breakage with instability, revision fixation needed. Seromas usually resolve with time |
| Tibial Tuberosity Avulsion |
Fracture of osteotomized fragment. Third most common major complication. Results from inadequate fragment size, insufficient fixation, or excessive quadriceps tension |
Immediate revision surgery with additional fixation. Lag screw and tension band wire. May require bone graft |
| Infection |
Uncommon (less than 5 percent). Risk factors: prolonged surgery time, excessive soft tissue trauma, postoperative seroma |
Culture and sensitivity. Appropriate antibiotics. May require implant removal if deep infection. Wound lavage |
| Progressive Osteoarthritis |
Expected to some degree in all cases. Severity depends on age at surgery, duration of luxation, and grade. Early surgery minimizes progression |
Long-term NSAIDs, joint supplements, weight management, physical rehabilitation. Polysulfated glycosaminoglycans may help |