NAVLE Musculoskeletal

Canine Fracture Management Study Guide

Fracture management is a fundamental component of veterinary orthopedic practice. Long bone fractures represent approximately 24% of all fractures in dogs, with the femur, tibia, radius/ulna, and humerus being most commonly affected.

Overview and Clinical Importance

Fracture management is a fundamental component of veterinary orthopedic practice. Long bone fractures represent approximately 24% of all fractures in dogs, with the femur, tibia, radius/ulna, and humerus being most commonly affected. Successful fracture management requires understanding of fracture classification, bone healing biology, fixation techniques, and recognition of complications. This knowledge is essential for the NAVLE examination and clinical practice.

The goal of fracture treatment is to restore anatomical alignment, maintain stability, promote bone healing, and return the patient to normal function while minimizing complications. Understanding when to use different fixation methods and recognizing when referral is appropriate are critical skills for the general practitioner.

Location Description and Clinical Significance
Diaphyseal Mid-shaft fractures; good blood supply; amenable to various fixation methods
Metaphyseal Flared ends near joints; cancellous bone; heals rapidly but challenging fixation
Epiphyseal End of bone; often involves articular surface; requires anatomic reduction
Physeal Growth plate fractures in immature animals; risk of growth disturbance
Articular Involves joint surface; requires perfect anatomic reduction to prevent arthritis

Fracture Classification Systems

Proper fracture classification is essential for communication between veterinarians, treatment planning, and prognosis determination. Fractures should be described systematically using multiple classification criteria.

Anatomic Classification

Fractures are first classified by anatomic location within the bone:

Fracture Configuration (Morphology)

High-YieldOn the NAVLE, remember that transverse fractures are the most stable configuration and may be amenable to IM pin fixation alone if well-reduced. Comminuted fractures are non-reconstructable and require bridging techniques (biological osteosynthesis) where implants bear the full load until healing occurs.
Configuration Description Stability
Transverse Perpendicular to long axis; right angle break Most stable; resists axial collapse when reduced
Oblique Diagonal across bone; angle less than 45 degrees Unstable; prone to shortening and rotation
Spiral Curved fracture line from torsional force Unstable; good candidate for cerclage wire
Comminuted Three or more fragments Very unstable; requires bridging fixation
Greenstick Incomplete fracture; one cortex intact Relatively stable; seen in young animals
Segmental Two or more separate fracture lines Highly unstable; complex fixation required

Salter-Harris Classification of Physeal Fractures

The Salter-Harris classification system is essential for describing growth plate fractures in skeletally immature dogs. Physeal injuries account for up to 30% of long bone fractures in young animals. The classification guides treatment and predicts prognosis for growth disturbance.

NAVLE TipRemember SALTR for Salter-Harris: S=Slip (I), A=Above (II), L=Lower (III), T=Through (IV), R=Rammed (V). Type II is most common (75%). Types III and IV are intra-articular and require anatomic reduction. Type V is often diagnosed RETROSPECTIVELY after growth arrest is noted. Never place screws across the physis in Types I and II as this causes premature closure!
Type Description Mnemonic (SALTR) Prognosis
Type I Fracture through physis only; epiphysis separates from metaphysis S = Slip (Straight across) Excellent
Type II Through physis and metaphysis; Thurston-Holland fragment A = Above (metaphysis) Good; MOST COMMON (75%)
Type III Through physis and epiphysis; intra-articular L = Lower (epiphysis) Fair; risk of arthritis
Type IV Through metaphysis, physis, AND epiphysis T = Through (everything) Guarded; requires perfect reduction
Type V Crush injury to physis; may not be visible on initial radiographs R = Rammed (crushed) POOR; high risk of premature closure

Open Fracture Classification: Gustilo-Anderson System

Open (compound) fractures involve disruption of skin integrity with communication between the fracture and external environment. These injuries carry significantly higher risk of infection, delayed healing, and complications. The Gustilo-Anderson classification guides treatment and predicts prognosis.

Open Fracture Management Principles

  • Antibiotics within 3 hours of injury - reduces infection rate 6-fold
  • Wound lavage with isotonic saline (minimum 1 liter per Type I; more for higher grades)
  • Aggressive surgical debridement of all nonviable tissue
  • Temporary or definitive fracture stabilization
  • Wound management (delayed primary closure often preferred for Type III)
  • External skeletal fixation is ideal - keeps implants away from contaminated zone
High-YieldOn NAVLE, if you see an open fracture with extensive soft tissue damage or a gunshot wound, think Type III Gustilo-Anderson. Early antibiotics (cephalosporin plus aminoglycoside for Type III) and thorough debridement are essential. The historical 6-hour rule for debridement is now considered less absolute, but prompt antibiotic administration remains critical.
Type Wound Characteristics Infection Rate and Management
Type I Wound less than 1 cm; clean; minimal soft tissue damage; often inside-out injury ~2% infection rate; 1st generation cephalosporin (cefazolin)
Type II Wound greater than 1 cm; moderate soft tissue damage; no extensive flaps or avulsions ~8% infection rate; 1st generation cephalosporin
Type IIIa Extensive soft tissue damage; high energy; adequate soft tissue coverage possible Add aminoglycoside (gentamicin) for gram-negative coverage
Type IIIb Extensive soft tissue loss; periosteal stripping; requires flap coverage High infection risk (greater than 40%); aggressive debridement
Type IIIc Associated vascular injury requiring repair; rare in small animals Limb-threatening; may require amputation

Bone Healing Biology

Understanding the two types of bone healing is crucial for selecting appropriate fixation methods and monitoring fracture repair progress.

Direct (Primary) Bone Healing

Direct healing occurs with rigid fixation and anatomic reduction resulting in a fracture gap less than 1mm and interfragmentary strain less than 2%. This is achieved with compression plating and lag screws.

  • Contact healing: Gap less than 0.01mm; direct osteonal remodeling across fracture line
  • Gap healing: Gap less than 1mm; lamellar bone fills gap followed by Haversian remodeling
  • No callus formation: Fracture line gradually disappears on radiographs
  • Timeline: Slower process; implants typically remain 5-14 months

Indirect (Secondary) Bone Healing

Indirect healing is the most common type in veterinary patients and occurs with less rigid fixation (casts, external fixators, IM pins). It involves callus formation through three overlapping phases:

Radiographic Assessment of Bone Healing

Clinical union is confirmed when callus bridges the fracture on at least 3 of 4 cortices visible on orthogonal radiographic views.

  • 5-7 days: Fracture gap widening and smudging of fracture edges (resorption phase)
  • 2-3 weeks: Early periosteal callus visible
  • 4-8 weeks: Callus becoming more radiopaque; fracture line becoming less distinct
  • 8-16 weeks: Bridging callus; clinical union in most adult dogs
Phase Timeline Key Events
Inflammatory 0-4 days Hematoma formation; inflammatory cells release growth factors; mesenchymal stem cell recruitment
Reparative 4 days to weeks Soft callus (fibrocartilage) forms; then mineralized hard callus via endochondral ossification; woven bone bridges fracture
Remodeling Months to years Woven bone replaced by lamellar bone; callus resorbed; bone shape restored per Wolff's Law

Fracture Fixation Methods

Selection of fixation method depends on fracture configuration, patient factors, and the forces that must be neutralized. Understanding the biomechanical properties of each method is essential.

Forces Acting on Fractures

Fixation Methods Comparison

High-YieldNAVLE loves to test the TWO MOST COMMON FRACTURE REPAIR ERRORS: (1) Using IM pins alone for radius fractures - the narrow canal prevents adequate pin size, and pins cannot resist rotation. ALWAYS use a plate for radius/ulna fractures. (2) Using IM pin and cerclage wire for NON-RECONSTRUCTABLE comminuted fractures - cerclage requires perfect anatomic reconstruction to function. Use bridging techniques (plate-rod, ESF, ILN) instead.

The 50/50 Rule

Fracture ends should have at least 50% cortical contact for bone healing to occur. If anatomic reduction cannot achieve 50% contact, internal fixation is required. This is the absolute minimum for healing to be possible (not probable).

Force Description and Fixation Considerations
Bending Force that angulates the bone; well resisted by IM pins and bone plates
Rotation (Torsion) Twisting force; IM pins CANNOT resist; requires supplemental fixation (cerclage, ESF, plate)
Axial Compression Force causing shortening; requires bony architecture (reduced transverse fracture) or load-sharing implant
Tension Pulling force; seen at muscle/tendon insertions; requires tension band technique
Shear Sliding force across fracture; requires interfragmentary compression (lag screw)

Fracture Healing Complications

Delayed Union

Delayed union is diagnosed when a fracture is taking longer to heal than expected for a fracture of that type, in a patient of similar age and health, with similar fixation. Healing activity is still present but slower than normal.

Causes:

  • Inadequate immobilization or stability
  • Excessive fracture gap or distraction
  • Impaired blood supply
  • Infection
  • Excessive implant material interfering with biology

Nonunion

Nonunion is a fracture where all osteogenic activity has ceased and healing will not occur without intervention. Generally diagnosed after 3 months or when no progression is seen on 2-3 consecutive radiographic evaluations.

Osteomyelitis

Osteomyelitis is bacterial infection of bone, most commonly posttraumatic (open fractures) or postsurgical in veterinary patients. It is the most common cause of nonunion when associated with infection.

Risk factors:

  • Open fractures with significant contamination
  • Presence of implants (biofilm formation)
  • Extensive soft tissue trauma
  • Necrotic tissue, hematoma, poor vascularity
  • Fracture instability

Radiographic signs: Excessive periosteal reaction, radiolucency around implants (loosening), sequestra, involucrum, draining tracts.

Treatment principle: Infected fractures CAN heal if they are STABLE. Treatment involves debridement, culture-based antibiotic therapy (long-term), and adequate stabilization. External fixation is ideal. Implant removal once clinical union is achieved.

Malunion

Malunion occurs when a fracture heals in abnormal alignment (angulation, rotation, or shortening). May cause lameness, abnormal joint loading, and degenerative joint disease. Corrective osteotomy may be required if functional impairment is significant.

NAVLE TipFor NAVLE, remember the difference between nonunion types: HYPERTROPHIC = Horse Shoe (lots of callus, looks like an elephant foot, just needs stability). ATROPHIC = Absent callus (avascular, needs graft and debridement). The key question is: Is the tissue biologically active? If yes (hypertrophic), stabilize. If no (atrophic), revascularize and graft.
Method Best Indications Key Considerations
External Coaptation Minimally displaced fractures below elbow/stifle in young dogs; greenstick fractures Must immobilize joint above and below; high complication rate; monitor weekly
IM Pin + Cerclage Long oblique/spiral RECONSTRUCTABLE fractures of femur, humerus, tibia Pin fills 60-70% canal; cerclage requires perfect reconstruction; AVOID for radius
External Skeletal Fixation Open fractures; comminuted fractures; infected fractures; radius/ulna; tibia Keeps implants away from fracture; allows wound access; requires client compliance
Bone Plate Most fracture types; articular fractures; radius/ulna in small breeds; mandibular fractures Compression, neutralization, bridging, or buttress function; gold standard for many fractures
Locking Plate Osteoporotic bone; metaphyseal fractures; comminuted fractures; short juxta-articular segments Screws lock to plate creating fixed-angle construct; less reliance on bone-screw interface
Interlocking Nail Femoral, humeral, tibial diaphyseal fractures; comminuted mid-diaphyseal fractures Resists all forces; load-bearing; biological technique preserves vascularity

Special Clinical Considerations

Radius/Ulna Fractures in Toy Breeds

Distal radius/ulna fractures in toy and miniature breeds have the highest complication rate of any long bone fracture. This is due to poor intraosseous blood supply in the distal radius of small dogs, minimal soft tissue coverage, and tendency for delayed union and nonunion. NEVER use IM pins for these fractures. Bone plate fixation is the treatment of choice, often with bone grafting.

Fracture Disease

Fracture disease encompasses complications that affect limb function after fracture healing, including muscle atrophy, reduced range of motion, osteoporosis, and quadriceps contracture (tie-down). Quadriceps contracture is especially associated with distal femoral fractures in young dogs. Prevention requires early passive range of motion exercises, adequate analgesia, and avoiding prolonged immobilization.

Type Radiographic Features Treatment Approach
Hypertrophic (Viable) Abundant callus; elephant foot appearance; blood supply intact Rigid stabilization alone often sufficient; bone graft not required
Oligotrophic (Viable) Little to no callus; blood supply present but not stimulated Rigid stabilization plus bone graft
Atrophic (Nonviable) No callus; rounded, resorbed bone ends; avascular Debridement; cancellous bone graft; rigid fixation; may require en bloc ostectomy

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